The Concise Encyclopedia of Fibromyalgia and Myofascial Pain

ISBN-13: 9780789015280 / Angielski / Miękka / 2002 / 324 str.

Roberto Patarca-Montero

Opis

Compiled by one of the foremost researchers in the field of immunology, The Concise Encyclopedia of Fibromyalgia and Myofascial Pain is an A-to-Z summary of current knowledge that updates patients and health care professionals on these disabling clinical disorders. This vital information has been organized in an easy-to-use format that lets you become familiar with highlights of the most relevant topics.

Spis treści

• Foreword
• Preface
• Chapter 1. Molecular Recognition Processes Between Plant and Bacterial Pathogens
• Introduction
• Physical Contact of Plant Cells is Necessary for Bacterial Recognition
• Molecules Responsible for Physical Contact
• Many Bacterial Pathogens Induce Necrosis on Hosts and Nonhosts
• Bacterial Pathogens Grow in Both Host and Nonhost Plants
• Bacterial Pathogens Induce Leakage of Nutrients in Both Host and Nonhost Plants
• Bacterial Genes Involved in Recognition of Hosts and Nonhosts
• Coregulation of hrp, avr and Other Pathogenicity Genes
• Transcription of Bacterial Pathogenicity Genes in Planta
• Plant-Derived Molecules May Be Involved in Induction of Bacterial Genes
• Some Plant Signals May Direct Synthesis of Elicitors
• Secretion of Elicitors From Bacterial Cells in Plants
• The Role of hrp and avr Genes in Early Recognition Process in Plant-Bacterial Pathogen Interactions
• Other Signal Molecules of Bacterial Pathogens
• The Signal Transduction System
• Systemic Signal Induction
• Is Cell Death Involved in Signal Transduction Pathway?
• How Pathogens Avoid or Overcome Host Defense Mechanisms Induced by the Signal Transduction System?
• Possible Role of Signal Transduction System in Evasion of Host Recognition by Phytopathogenic Bacteria During Pathogenesis
• Conclusion
• Chapter 2. Host Defense Mechanisms: Cell Wall?the First Barrier and a Source of Defense Signal Molecules
• The First Barrier to Bacterial Infection in Plants
• Structure of the Plant Cell Wall
• Pectic Polysaccharides
• Cellulose
• Hemicellulos
• Cell Wall Proteins
• Bacterial Genes Encoding Extracellular Enzymes
• Bacterial Genes Regulating Production of Extracellular Enzymes
• Bacterial Genes Regulating Secretion of Extracellular Enzymes
• Secretion of Proteases
• The Signaling System in Induction of Bacterial Extracellular Enzymes
• Plant Cell Wall Components Involved in Defense Mechanisms Against Bacterial Pathogens
• Bacterial Extracellular Enzymes Induce Host Defense Mechanisms
• Pectic Fragments Induce Virulence Genes in Bacterial and Defense Genes in Plants
• Pectic Enzymes Vary in Inducing Resistance or Susceptibility
• Polygalacturonase-Inhibiting Proteins
• Cell Wall Modifications and Bacterial Disease Resistance
• Conclusion
• Chapter 3. Active Oxygen Species
• Mechanism of Production of Active Oxygen Species
• Signals for Induction of Active Oxygen Species in Bacteria-Infected Plants
• Bacterial Infection Leads to Production of Active Oxygen Species in Plants
• Active Oxygen Species May Induce Lipid Peroxidation
• Increases in Active Oxygen Species Lead to Activation of Lipoxygenase
• Active Oxygen Species Production Leads to Cell Membrane Damage
• Active Oxygen Species May Directly Kill Bacterial Pathogens
• Bacterial Pathogens May Tolerate Toxicity of Active Oxygen Species
• Antioxidants of the Host May Protect Bacterial Pathogens Against Active Oxygen Species
• The Possible Role of Active Oxygen Species in Disease Resistance
• Conclusion
• Chapter 4. Inducible Plant Proteins
• Introduction
• Nomenclature of Pathogen-Inducible Plant Proteins
• Occurrence of PR Proteins in Various Plants
• Classification of PR Proteins
• Bacterial Pathogens Induce PR Proteins
• Molecular Mechanisms of Induction of PR Proteins
• Compartmentalization of PR Proteins in Plant Tissues
• The Role of PR Proteins in Bacterial Disease Resistance
• The Second Group of Pathogen-Inducible Proteins: Constitutive, but Increasingly Induced
• Hydroxyproline-Rich Glycoproteins
• Lectins
• Not All Inducible Proteins Need Be Involved in Inducing Bacterial Disease Resistance
• Conclusion
• Chapter 5. Inducible Secondary Metabolites
• What Are Inducible Secondary Metabolites?
• Bacterial Pathogens Induce Accumulation of Secondary Metabolites in Infected Tissues
• Phytoalexins Accumulate in Plants After Irreversible Cell Membrane Damage
• Phytoalexins Accumulate Only Locally and Not Systemically
• Mode of Syntheses of Phytoalexins
• Evidences That Induced Secondary Metabolites Are Involved in Bacterial Disease Resistance
• Phytoalexins May Be Suppressed, Degraded, or Inactivated in Susceptible Interactions
• Some Phytoalexins May Not Have Any Role in Disease Resistance
• Constitutive, but Induced Secondary Metabolites During Pathogenesis
• Conclusion
• Chapter 6. Biotechnological Applications: Molecular Manipulation of Bacterial Disease Resistance
• Introduction
• Manipulation of Signal Transduction System for Induction of Disease Resistance
• Manipulation of Resistance Genes Involved in Signal Transduction System
• Manipulation of Signal Transduction System by Elicitors
• Manipulation of Signal Transduction System by Using Chemicals
• Manipulation of Signal Transduction System by Using Rhizobacterial Strains
• Manipulation of Signal Transduction System by Enhanced Biosynthesis of Salicylic Acid
• Manipulation of Signal Transduction System by Inducing Accelerated Cell Death
• Manipulation of Signal Transduction System by Enhanced Biosynthesis of Cytokinins
• Manipulation of Inducible Proteins for Induction of Bacterial Disease Resistance
• Suppression of Virulence Factors of Bacterial Pathogens to Manage Bacterial Diseases
• Exploitation of Insect Genes Encoding Antibacterial Proteins for Bacterial Disease Management
• Exploitation of Bacteriophage Genes for Bacterial Disease Management
• Exploitation of Genes from Human Beings, Hens, and Crabs for Management of Plant Bacterial Diseases
• Conclusion
• References
• Index

O autorze

Roberto Patarca Montero